In the context of Graves' disease, why might beta-blockers be prescribed?

In Graves' disease, which is characterized by hyperthyroidism, patients often experience symptoms such as anxiety, palpitations, tremors, and increased heart rate due to elevated levels of thyroid hormones. Beta-blockers are effective in managing these symptoms by blocking the effects of adrenaline and other stress hormones, thereby reducing heart rate and alleviating anxiety. They do not act on the thyroid hormone production directly but rather help control the physiological responses associated with hyperthyroidism.

While the other options might sound relevant, they do not address the primary usage of beta-blockers in this context. For instance, beta-blockers do not enhance metabolic rates; rather, they help mitigate the heightened metabolic rate caused by excess thyroid hormones. They also do not directly reduce thyroid hormone levels or promote sleep as their primary function is to manage cardiovascular and peripheral symptoms of hyperthyroidism. Thus, the most appropriate reason for prescribing beta-blockers in Graves' disease is to manage symptoms of anxiety and the hyperthyroid state.